Matamalas, Joan
Dr. Joan T. Matamalas obtained his B.E. in Computer Science from the Open University of Catalonia, where he also received his Engineer’s degree in Computer Science. He went on to get his M.S. in Artificial Intelligence at the Polytechnic University of Catalonia. His PhD was obtained from the Rovira i Virgili University (Tarragona, Spain) under the supervision of Professors Alex Arenas and Sergio Gomez, where he worked on the study of higher-order dynamics on complex networks. He has published his findings in prestigious interdisciplinary peer-reviewed journals and participated in numerous international congresses.
Dr. Matamalas has been appointed Research Associate at Harvard Medical School and Research Scientist at Brigham and Women’s Hospital and works in Professor Masanori Aikawa’s research group. His current research focuses on the modeling of the complex heterogeneity among CAD patients. He is also involved in the mathematical modeling of COVID-19, covering different vital aspects, including its spreading, effect of containment policies, or the clinical outcome of those infected by the virus.
Publications
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Matamalas JT, Chelvanambi S, Decano JL, França RF, Halu A, Santinelli-Pestana DV, Aikawa E, Malhotra R, Aikawa M
Obesity and age are transmission risk factors for SARS-CoV-2 infection among exposed individuals.
Decano JL, Maiorino E, Matamalas JT, Chelvanambi S, Tiemeijer BM, Yanagihara Y, Mukai S, Jha PK, Pestana DVS, D'Souza E, Whelan M, Ge R, Asano T, Sharma A, Libby P, Singh SA, Aikawa E, Aikawa M
Cellular Heterogeneity of Activated Primary Human Macrophages and Associated Drug-Gene Networks: From Biology to Precision Therapeutics.
Decano JL, Iwamoto Y, Goto S, Lee JY, Matamalas JT, Halu A, Blaser M, Lee LH, Pieper B, Chelvanambi S, Silva-Nicolau J, Bartoli-Leonard F, Higashi H, Shibata H, Vyas P, Wang J, Gostjeva E, Body SC, Singh SA, Aikawa M, Aikawa E
A disease-driver population within interstitial cells of human calcific aortic valves identified via single-cell and proteomic profiling.
Iwata H, Osborn EA, Ughi GJ, Murakami K, Goettsch C, Hutcheson JD, Mauskapf A, Mattson PC, Libby P, Singh SA, Matamalas J, Aikawa E, Tearney GJ, Aikawa M, Jaffer FA
Highly Selective PPARα (Peroxisome Proliferator-Activated Receptor α) Agonist Pemafibrate Inhibits Stent Inflammation and Restenosis Assessed by Multimodality Molecular-Microstructural Imaging.
Decano JL, Singh SA, Bueno CG, Lee LH, Halu A, Chelvanambi S, Matamalas JT, Zhang H, Mlynarchik AK, Qiao J, Sharma A, Mukai S, Wang J, Anderson DG, Ozaki CK, Libby P, Aikawa E, Aikawa M
Systems Approach to Discovery of Therapeutic Targets for Vein Graft Disease PPARα Pivotally Regulates Metabolism, Activation, and Heterogeneity of Macrophages and Lesion Development.
